PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27816613-4	2017	By using morphological examination, flow cytometry, and mitochondrial membrane potential (DeltaYm) measurement, we confirmed that exposure to TDCIPP caused apoptosis accompanied by the activation of apoptosis-related genes (e.g. Bax and Bcl-2) and caspase 3 protein in SH-SY5Y cells.	tris(1,3-dichloro-2-propyl)phosphate	142-148	BCL2 associated X, apoptosis regulator	Homo sapiens	229-232	
32304943-6	2020	In addition, TDCPP induced cell apoptosis and arrested cell cycle in the G0/G1 phase at 16 and 160 mug/mL by enhancing Bax and Caspase-3 expression besides inhibiting cyclin D1, CDK2, CDK6 and Bcl-2 expression.	tris(1,3-dichloro-2-propyl)phosphate	13-18	BCL2 associated X, apoptosis regulator	Homo sapiens	119-122	
27816613-8	2017	Treatment with PBA effectively attenuated TDCIPP-induced ER stress and protected against apoptotic death in SH-SY5Y cells by inhibition of Bax expression and promotion of Bcl-2 expression.	tris(1,3-dichloro-2-propyl)phosphate	42-48	BCL2 associated X, apoptosis regulator	Homo sapiens	139-142