PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28224698-9	2017	In conclusion, CYP3A5 genotype and several clinical variables were identified as modulators of the tacrolimus-azole interaction, but these did not permit accurate predictions in individual patients.	Azoles	110-115	cytochrome P450 family 3 subfamily A member 5	Homo sapiens	15-21	
18220568-7	2008	Inhibitory effects of azole antifungal agents and macrolide antibiotics, with low Ki and/or IC50 values for CYP3A4, are likely to be determinant factors for predominant drug interactions in humans, although Asian subjects with relatively high frequency of genetic CYP3A5 expressers should be carefully treated with CYP3A substrates.	Azoles	22-27	cytochrome P450 family 3 subfamily A member 5	Homo sapiens	264-270