PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
1380512-6	1992	The induction of the CEA responses by TGF-beta 1 was also blocked by the protein kinase inhibitor 1-(isoquinolinesulfonyl)-2-methylpiperazine dihydrochloride (H-7), which also inhibited cellular PKC activity.	1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine	159-162	transforming growth factor beta 1	Homo sapiens	38-48	
8318816-4	1993	TGF-beta 1-induced IANK cell chemotaxis was inhibited by protein kinase C inhibitors, such as staurosporine and H7.	1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine	112-114	transforming growth factor beta 1	Homo sapiens	0-10	
1934283-6	1991	The protein kinase C (PKC) inhibitor H-7 induced a dose-dependent enhancement in communication, which was even higher in the presence of TGF-beta (4 pM X 24 h).	1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine	37-40	transforming growth factor beta 1	Homo sapiens	137-145	
12118074-2	2002	We found that TGFbeta-induced EMT was blocked by inhibiting activation of p38 mitogen-activated protein kinase (MAPK) with H-7, a protein kinase C inhibitor, and with SB202190, a direct inhibitor of p38MAPK.	1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine	123-126	transforming growth factor beta 1	Homo sapiens	14-21