PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 29055264-8 2018 Paroxetine treatment increased the levels of proinflammatory cytokines IFN-gamma, TNF-alpha, and IL-6 and decreased Th2 cytokine levels. Paroxetine 0-10 interleukin 6 Homo sapiens 97-101 29055264-9 2018 After paroxetine treatment, IL-6 levels increased more in the non-remitter group than in the remitter group. Paroxetine 6-16 interleukin 6 Homo sapiens 28-32 31375659-8 2019 Response to paroxetine treatment correlated with baseline IL-10, IL-6 and TNF-alpha levels, with the strongest signal being observed in males. Paroxetine 12-22 interleukin 6 Homo sapiens 65-69 19179025-8 2009 Serum IL-6 concentration in women treated with paroxetine decreased significantly. Paroxetine 47-57 interleukin 6 Homo sapiens 6-10 19179025-11 2009 CONCLUSION: Decrease in IL-6 concentration may be involved in the mechanism of the actions of both paroxetine and kamishoyosan in women with psychological symptoms, and IL-6 may therefore be useful as a marker of treatment. Paroxetine 99-109 interleukin 6 Homo sapiens 24-28 33545432-9 2021 These results suggest that plasma IL-6 level may be a promising biological marker for predicting the likely treatment response to paroxetine in individual patients with MDD. Paroxetine 130-140 interleukin 6 Homo sapiens 34-38 33602262-7 2021 RESULTS: All the SSRIs, including paroxetine, fluoxetine, sertraline, citalopram, and fluvoxamine, show a visible cytotoxicity within the range of applied doses, and a paradoxical effect on astrocytic inflammatory responses as manifested by the promotion of inducible nitric oxide synthase (iNOS) and/or nitric oxide (NO) and the inhibition of interleukin 6 (IL-6) and/or interleukin 1beta (IL-1beta). Paroxetine 34-44 interleukin 6 Homo sapiens 344-357 33602262-7 2021 RESULTS: All the SSRIs, including paroxetine, fluoxetine, sertraline, citalopram, and fluvoxamine, show a visible cytotoxicity within the range of applied doses, and a paradoxical effect on astrocytic inflammatory responses as manifested by the promotion of inducible nitric oxide synthase (iNOS) and/or nitric oxide (NO) and the inhibition of interleukin 6 (IL-6) and/or interleukin 1beta (IL-1beta). Paroxetine 34-44 interleukin 6 Homo sapiens 359-363