PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32953930-6	2020	Because Smad7 functions as an inhibitor for both TGF-beta/Smad and nuclear factor kappaB (NF-kappaB) signaling, increased cardiac Smad7 could be another mechanism through which SIS3 treatment blocked Smad3-mediated myocardial fibrosis and NF-kappaB-driven cardiac inflammation.	6,7-dimethyl-2-(2E)-3-(1-methyl-2-phenyl-1H-pyrrolo(2,3-b)pyridin-3-yl-prop-2-enoyl)-1,2,3,4-tetrahydroisoquinoline hydrochloride	177-181	SMAD family member 7	Mus musculus	8-13	
32953930-6	2020	Because Smad7 functions as an inhibitor for both TGF-beta/Smad and nuclear factor kappaB (NF-kappaB) signaling, increased cardiac Smad7 could be another mechanism through which SIS3 treatment blocked Smad3-mediated myocardial fibrosis and NF-kappaB-driven cardiac inflammation.	6,7-dimethyl-2-(2E)-3-(1-methyl-2-phenyl-1H-pyrrolo(2,3-b)pyridin-3-yl-prop-2-enoyl)-1,2,3,4-tetrahydroisoquinoline hydrochloride	177-181	SMAD family member 7	Mus musculus	8-12	
32953930-6	2020	Because Smad7 functions as an inhibitor for both TGF-beta/Smad and nuclear factor kappaB (NF-kappaB) signaling, increased cardiac Smad7 could be another mechanism through which SIS3 treatment blocked Smad3-mediated myocardial fibrosis and NF-kappaB-driven cardiac inflammation.	6,7-dimethyl-2-(2E)-3-(1-methyl-2-phenyl-1H-pyrrolo(2,3-b)pyridin-3-yl-prop-2-enoyl)-1,2,3,4-tetrahydroisoquinoline hydrochloride	177-181	SMAD family member 7	Mus musculus	130-135