PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26683309-6	2016	In vitro, cells treated with T-2/HT-2 showed reductions of GSH concentration and significant increases in LDH leakage, ALT/AST ROS, GSH-PX, SOD and CAT activities, MDA concentration, and expression of mRNA related to oxidative stress.	ros	127-130	solute carrier family 25 member 5	Homo sapiens	29-32	
22429830-7	2013	When combined, ZEN and T-2 toxin increased ROS production and heat shock protein (Hsp) 70 expression as compared to the effect of each mycotoxin taken alone.	ros	43-46	solute carrier family 25 member 5	Homo sapiens	23-26	
25264878-5	2014	Exposure to T-2 toxin can reduce activities of mitochondrial complexes III, IV and V, DeltaPsim and the cellular ATP, while intracellular ROS increased following treatment with T-2 toxin.	ros	138-141	solute carrier family 25 member 5	Homo sapiens	177-180	
24030954-7	2013	Further cellular analysis showed that T-2 toxin was able to induce the ROS accumulation and could lead to an increase in mitochondrial mass and adenosine 5"-triphosphate content, which indicated that oxidative stress and mitochondrial enhancement occurred in T-2 toxin-treated cells.	ros	71-74	solute carrier family 25 member 5	Homo sapiens	38-41	
25917637-9	2015	Furthermore, up-regulation of NOD2 expression induced by T-2 toxin could be abrogated by pretreating the cells with inhibitors of NF-kappaB and scavenger of ROS.	ros	157-160	solute carrier family 25 member 5	Homo sapiens	57-60	
25917637-10	2015	CONCLUSION: T-2 toxin could up-regulate NOD2 expression via ROS/NF-kappaB pathway and activate NOD2 signaling pathway.	ros	60-63	solute carrier family 25 member 5	Homo sapiens	12-15