PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34330964-3	2021	Despite their efficacy in reactivating AChE, the action of drugs like 2-pralidoxime (2-PAM) is primarily limited to the peripheral nervous system (PNS) and, thus, provides no significant protection to the central nervous system (CNS).	Pralidoxime Compounds	85-90	acetylcholinesterase (Cartwright blood group)	Homo sapiens	39-43	
16769211-7	2006	The sensor remained 94% of its original activity after six cycles of inhibition with 500 ppb paraoxon followed with reactivation of AChE by 0.5 mM 2-pyriding-aldoxime methoiodide (2-PAM).	Pralidoxime Compounds	180-185	acetylcholinesterase (Cartwright blood group)	Homo sapiens	132-136	
31879781-5	2020	The primary therapeutic strategy employed in the U.S. to treat OP intoxication includes reactivation of inhibited AChE with the oxime pralidoxime (2-PAM) along with the muscarinic acetylcholine receptor antagonist atropine and the benzodiazepine, diazepam.	Pralidoxime Compounds	147-152	acetylcholinesterase (Cartwright blood group)	Homo sapiens	114-118	
23111374-6	2013	Regarding the reactivation of VX-inhibited hAChE, all compounds show a better reactivation potency than those of 2-PAM, nevertheless they are less efficient than obidoxime and HI-6.	Pralidoxime Compounds	113-118	acetylcholinesterase (Cartwright blood group)	Homo sapiens	43-48	
23111374-7	2013	Moreover, one of seven described compounds presents an ability to reactivate tabun-inhibited hAChE equivalent to those of 2-PAM.	Pralidoxime Compounds	122-127	acetylcholinesterase (Cartwright blood group)	Homo sapiens	93-98