PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 10397596-4 1999 At lower concentration, phenolic antioxidants and isothiocyanates activate mitogen-activated protein kinase (MAPK; extracellular signal-regulated protein kinase 2, ERK2; and c-Jun N-terminal kinase 1, JNK1) in a concentration-and time-dependent manner. Isothiocyanates 50-65 mitogen-activated protein kinase 1 Homo sapiens 109-113 10397596-4 1999 At lower concentration, phenolic antioxidants and isothiocyanates activate mitogen-activated protein kinase (MAPK; extracellular signal-regulated protein kinase 2, ERK2; and c-Jun N-terminal kinase 1, JNK1) in a concentration-and time-dependent manner. Isothiocyanates 50-65 mitogen-activated protein kinase 1 Homo sapiens 164-168 28969043-10 2017 Thus, isothiocyanates act as anti-metastatic compounds that suppress MMP-9 activity/expression by inhibiting NF-kappaB and AP-1 via suppression of the FAK/ERK and FAK/Akt signaling pathways. Isothiocyanates 6-21 mitogen-activated protein kinase 1 Homo sapiens 155-158 16272172-0 2006 ERK and JNK signaling pathways are involved in the regulation of activator protein 1 and cell death elicited by three isothiocyanates in human prostate cancer PC-3 cells. Isothiocyanates 118-133 mitogen-activated protein kinase 1 Homo sapiens 0-3 16928811-0 2006 Mechanism of action of isothiocyanates: the induction of ARE-regulated genes is associated with activation of ERK and JNK and the phosphorylation and nuclear translocation of Nrf2. Isothiocyanates 23-38 mitogen-activated protein kinase 1 Homo sapiens 110-113