PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10397596-4	1999	At lower concentration, phenolic antioxidants and isothiocyanates activate mitogen-activated protein kinase (MAPK; extracellular signal-regulated protein kinase 2, ERK2; and c-Jun N-terminal kinase 1, JNK1) in a concentration-and time-dependent manner.	Isothiocyanates	50-65	mitogen-activated protein kinase 1	Homo sapiens	109-113	
10397596-4	1999	At lower concentration, phenolic antioxidants and isothiocyanates activate mitogen-activated protein kinase (MAPK; extracellular signal-regulated protein kinase 2, ERK2; and c-Jun N-terminal kinase 1, JNK1) in a concentration-and time-dependent manner.	Isothiocyanates	50-65	mitogen-activated protein kinase 1	Homo sapiens	164-168	
28969043-10	2017	Thus, isothiocyanates act as anti-metastatic compounds that suppress MMP-9 activity/expression by inhibiting NF-kappaB and AP-1 via suppression of the FAK/ERK and FAK/Akt signaling pathways.	Isothiocyanates	6-21	mitogen-activated protein kinase 1	Homo sapiens	155-158	
16272172-0	2006	ERK and JNK signaling pathways are involved in the regulation of activator protein 1 and cell death elicited by three isothiocyanates in human prostate cancer PC-3 cells.	Isothiocyanates	118-133	mitogen-activated protein kinase 1	Homo sapiens	0-3	
16928811-0	2006	Mechanism of action of isothiocyanates: the induction of ARE-regulated genes is associated with activation of ERK and JNK and the phosphorylation and nuclear translocation of Nrf2.	Isothiocyanates	23-38	mitogen-activated protein kinase 1	Homo sapiens	110-113