Title : Treatment of multiple sclerosis with copolymer-1 (Copaxone): implicating mechanisms of Th1 to Th2/Th3 immune-deviation.

Pub. Date : 1998 Dec 1

PMID : 9916886






3 Functional Relationships(s)
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1 Treatment of multiple sclerosis with copolymer-1 (Copaxone): implicating mechanisms of Th1 to Th2/Th3 immune-deviation. Glatiramer Acetate negative elongation factor complex member C/D Homo sapiens
2 Treatment of multiple sclerosis with copolymer-1 (Copaxone): implicating mechanisms of Th1 to Th2/Th3 immune-deviation. Glatiramer Acetate negative elongation factor complex member C/D Homo sapiens
3 These results suggest that the beneficial clinical effects of Copaxone in MS patients may be attributed to changes in activation of T cell subsets and a shift from Th1 to Th2/Th3 cytokine profile, probably leading to Cop-1-driven mechanisms of bystander suppression. Glatiramer Acetate negative elongation factor complex member C/D Homo sapiens