Title : Transient stimulation of the c-Jun-NH2-terminal kinase/activator protein 1 pathway and inhibition of extracellular signal-regulated kinase are early effects in paclitaxel-mediated apoptosis in human B lymphoblasts.

Pub. Date : 1998 Jan 15

PMID : 9443400






2 Functional Relationships(s)
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Sentence
Compound Name
Protein Name
Organism
1 Paclitaxel caused a rapid and transient increase in c-Jun NH2-terminal kinase (JNK) activity, a proposed mediator of stress activation pathways. Paclitaxel mitogen-activated protein kinase 8 Homo sapiens
2 Paclitaxel caused a rapid and transient increase in c-Jun NH2-terminal kinase (JNK) activity, a proposed mediator of stress activation pathways. Paclitaxel mitogen-activated protein kinase 8 Homo sapiens