Pub. Date : 1997 Jun 3
PMID : 9177459
5 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
| 1 | Modulation of apoptosis through altered expression of Bcl-2 and/or Bax may be a mechanism by which dehydroepiandrosterone (DHEA) administration and calorie restriction (CR) exert their chemopreventive effects in p53-deficient (p53-/-) mice. | Dehydroepiandrosterone | transformation related protein 53, pseudogene | Mus musculus |
| 2 | Modulation of apoptosis through altered expression of Bcl-2 and/or Bax may be a mechanism by which dehydroepiandrosterone (DHEA) administration and calorie restriction (CR) exert their chemopreventive effects in p53-deficient (p53-/-) mice. | Dehydroepiandrosterone | transformation related protein 53, pseudogene | Mus musculus |
| 3 | Modulation of apoptosis through altered expression of Bcl-2 and/or Bax may be a mechanism by which dehydroepiandrosterone (DHEA) administration and calorie restriction (CR) exert their chemopreventive effects in p53-deficient (p53-/-) mice. | Dehydroepiandrosterone | transformation related protein 53, pseudogene | Mus musculus |
| 4 | Modulation of apoptosis through altered expression of Bcl-2 and/or Bax may be a mechanism by which dehydroepiandrosterone (DHEA) administration and calorie restriction (CR) exert their chemopreventive effects in p53-deficient (p53-/-) mice. | Dehydroepiandrosterone | transformation related protein 53, pseudogene | Mus musculus |
| 5 | Treatment of p53-/- animals with DHEA resulted in decreased Bcl-2 but not Bax mRNA levels in the thymus. | Dehydroepiandrosterone | transformation related protein 53, pseudogene | Mus musculus |