Title : Mutation of an aspartate at position 63 in the human platelet-activating factor receptor augments binding affinity but abolishes G-protein-coupling and inositol phosphate production.

Pub. Date : 1996 Feb 27

PMID : 8645288






2 Functional Relationships(s)
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1 Mutation of an aspartate at position 63 in the human platelet-activating factor receptor augments binding affinity but abolishes G-protein-coupling and inositol phosphate production. Aspartic Acid platelet activating factor receptor Homo sapiens
2 Mutation of an aspartate at position 63 in the human platelet-activating factor receptor augments binding affinity but abolishes G-protein-coupling and inositol phosphate production. Inositol Phosphates platelet activating factor receptor Homo sapiens