Title : NADPH oxidases and HIF1 promote cardiac dysfunction and pulmonary hypertension in response to glucocorticoid excess.

Pub. Date : 2020 Jul

PMID : 32413743






1 Functional Relationships(s)
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1 Thus, p22phox-dependent NADPH oxidases and activation of the HIF pathway are critical elements in dexamethasone-induced cardiovascular pathologies and might provide interesting targets to limit cardiovascular side effects in patients on chronic glucocorticoid therapy. Dexamethasone cytochrome b-245 alpha chain Homo sapiens