Title : Hypercholesterolemia blunts the oxidative stress elicited by hypertension in venules through angiotensin II type-2 receptors.

Pub. Date : 2016 May

PMID : 26775070






3 Functional Relationships(s)
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1 The inhibitory effects of HCh on the AngII mediated responses were also observed in genetically-induced HCh (ApoE-deficient mice). alpha-hexachlorocyclohexane apolipoprotein E Mus musculus
2 The inhibitory effects of HCh on the AngII mediated responses were also observed in genetically-induced HCh (ApoE-deficient mice). alpha-hexachlorocyclohexane apolipoprotein E Mus musculus
3 CONCLUSIONS: These findings indicate that HCh blunts the oxidative stress and inflammatory cell recruitment elicited by hypertension in venules through a mechanism that involves AT2 receptor activation. alpha-hexachlorocyclohexane serine (or cysteine) peptidase inhibitor, clade B, member 9d Mus musculus