Title : Some aspects of neurophysiological basis of insecticide action.

Pub. Date : 1976 Dec

PMID : 186331






7 Functional Relationships(s)
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1 These effects of parathion on the MES and evoked potentials coincided with a fall in blood and brain acetylcholinesterase (AChe) activities but disappeared after AChe inhibition had reached its peak and stabilized. Parathion acetylcholinesterase Rattus norvegicus
2 These effects of parathion on the MES and evoked potentials coincided with a fall in blood and brain acetylcholinesterase (AChe) activities but disappeared after AChe inhibition had reached its peak and stabilized. Parathion acetylcholinesterase Rattus norvegicus
3 These effects of parathion on the MES and evoked potentials coincided with a fall in blood and brain acetylcholinesterase (AChe) activities but disappeared after AChe inhibition had reached its peak and stabilized. Parathion acetylcholinesterase Rattus norvegicus
4 These effects of parathion on the MES and evoked potentials coincided with a fall in blood and brain acetylcholinesterase (AChe) activities but disappeared after AChe inhibition had reached its peak and stabilized. 2-(N-morpholino)ethanesulfonic acid acetylcholinesterase Rattus norvegicus
5 These effects of parathion on the MES and evoked potentials coincided with a fall in blood and brain acetylcholinesterase (AChe) activities but disappeared after AChe inhibition had reached its peak and stabilized. 2-(N-morpholino)ethanesulfonic acid acetylcholinesterase Rattus norvegicus
6 Because these measurements of central nervous system function returned to normal despite continued inhibition of AChe activity, the results are interpreted to mean either that adaptation of evoked potentials or MES responses to prolonged AChe inhibition can occur in the rat and monkey after parathion administration or that some of the effects of parathion do not depend on AChe inhibition. 2-(N-morpholino)ethanesulfonic acid acetylcholinesterase Rattus norvegicus
7 Because these measurements of central nervous system function returned to normal despite continued inhibition of AChe activity, the results are interpreted to mean either that adaptation of evoked potentials or MES responses to prolonged AChe inhibition can occur in the rat and monkey after parathion administration or that some of the effects of parathion do not depend on AChe inhibition. 2-(N-morpholino)ethanesulfonic acid acetylcholinesterase Rattus norvegicus