Title : GABA release and uptake regulate neuronal precursor migration in the postnatal subventricular zone.

Pub. Date : 2004 Sep 1

PMID : 15342728






4 Functional Relationships(s)
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1 Application of GABA at 10 microm, a nondesensitizing concentration for GABA(A) receptors (GABA(A)Rs), reduced the rate (mean of approximately 50 microm/hr) of cell migration by 21% via GABA(A)R activation. gamma-Aminobutyric Acid gamma-aminobutyric acid (GABA) A receptor, subunit gamma 2 Mus musculus
2 Application of the GABA(A)R antagonist bicuculline enhanced the migration rate by 30%, suggesting that endogenous GABA tonically reduces the speed of cell migration via GABA(A)R activation. Bicuculline gamma-aminobutyric acid (GABA) A receptor, subunit gamma 2 Mus musculus
3 Application of the GABA(A)R antagonist bicuculline enhanced the migration rate by 30%, suggesting that endogenous GABA tonically reduces the speed of cell migration via GABA(A)R activation. Bicuculline gamma-aminobutyric acid (GABA) A receptor, subunit gamma 2 Mus musculus
4 Application of the GABA(A)R antagonist bicuculline enhanced the migration rate by 30%, suggesting that endogenous GABA tonically reduces the speed of cell migration via GABA(A)R activation. gamma-Aminobutyric Acid gamma-aminobutyric acid (GABA) A receptor, subunit gamma 2 Mus musculus