Title : Role of phospholipase C, protein kinase C, and calcium in VEGF-induced venular hyperpermeability.

Pub. Date : 1999 Feb

PMID : 9950855






2 Functional Relationships(s)
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1 VEGF (10(-10) M) induced a two- to threefold increase in Pa, which was blocked by a monoclonal antibody directed against the VEGF receptor Flk-1/KDR, the phospholipase C (PLC) antagonist U-73122, or the protein kinase C (PKC) antagonist bisindolylmaleimide (BIM). Protactinium vascular endothelial growth factor A Homo sapiens
2 VEGF (10(-10) M) induced a two- to threefold increase in Pa, which was blocked by a monoclonal antibody directed against the VEGF receptor Flk-1/KDR, the phospholipase C (PLC) antagonist U-73122, or the protein kinase C (PKC) antagonist bisindolylmaleimide (BIM). Protactinium vascular endothelial growth factor A Homo sapiens