Title : Transfection of inducible nitric oxide synthase gene causes apoptosis in vascular smooth muscle cells.

Pub. Date : 1998 Sep 22

PMID : 9743513






4 Functional Relationships(s)
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Protein Name
Organism
1 METHODS AND RESULTS: VSMCs transiently transfected with iNOS cDNA functionally expressed 130 kd iNOS protein with full catalytic activity to generate massive NO in proportion to the doses of cDNA used; its enzymatic activity as well as NO production was completely blocked by an NOS inhibitor, NG-monomethyl-L-arginine (LNMMA). omega-N-Methylarginine nitric oxide synthase 2 Homo sapiens
2 METHODS AND RESULTS: VSMCs transiently transfected with iNOS cDNA functionally expressed 130 kd iNOS protein with full catalytic activity to generate massive NO in proportion to the doses of cDNA used; its enzymatic activity as well as NO production was completely blocked by an NOS inhibitor, NG-monomethyl-L-arginine (LNMMA). omega-N-Methylarginine nitric oxide synthase 2 Homo sapiens
3 METHODS AND RESULTS: VSMCs transiently transfected with iNOS cDNA functionally expressed 130 kd iNOS protein with full catalytic activity to generate massive NO in proportion to the doses of cDNA used; its enzymatic activity as well as NO production was completely blocked by an NOS inhibitor, NG-monomethyl-L-arginine (LNMMA). omega-N-Methylarginine nitric oxide synthase 2 Homo sapiens
4 METHODS AND RESULTS: VSMCs transiently transfected with iNOS cDNA functionally expressed 130 kd iNOS protein with full catalytic activity to generate massive NO in proportion to the doses of cDNA used; its enzymatic activity as well as NO production was completely blocked by an NOS inhibitor, NG-monomethyl-L-arginine (LNMMA). omega-N-Methylarginine nitric oxide synthase 2 Homo sapiens