Title : Resistance of human leukemic CEM-C1 cells is overcome by synergism between glucocorticoid and protein kinase A pathways: correlation with c-Myc suppression.

Pub. Date : 1998 Aug 15

PMID : 9721879






3 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 In these, forskolin (with or without Dex) caused a similar increase in cAMP (approximately 300-fold) and phospho-cAMP-responsive element binding protein (approximately 4-5-fold) levels, whereas total cAMP-responsive element binding protein expression was not affected. Colforsin cathelicidin antimicrobial peptide Homo sapiens
2 In these, forskolin (with or without Dex) caused a similar increase in cAMP (approximately 300-fold) and phospho-cAMP-responsive element binding protein (approximately 4-5-fold) levels, whereas total cAMP-responsive element binding protein expression was not affected. Colforsin cathelicidin antimicrobial peptide Homo sapiens
3 In these, forskolin (with or without Dex) caused a similar increase in cAMP (approximately 300-fold) and phospho-cAMP-responsive element binding protein (approximately 4-5-fold) levels, whereas total cAMP-responsive element binding protein expression was not affected. Colforsin cathelicidin antimicrobial peptide Homo sapiens