Title : The cellular response to oxidative stress: influences of mitogen-activated protein kinase signalling pathways on cell survival.

Pub. Date : 1998 Jul 15

PMID : 9657968






2 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 Overexpression of the anti-apoptotic protein Bcl-2 significantly decreased the apoptosis seen after treatment with H2O2 without altering ERK or JNK/SAPK activities. Hydrogen Peroxide BCL2 apoptosis regulator Homo sapiens
2 Bcl-2 acts independently and downstream of ERK and JNK/SAPK to enhance the survival of H2O2-treated cells. Hydrogen Peroxide BCL2 apoptosis regulator Homo sapiens