Pub. Date : 1998 Mar
PMID : 9576062
6 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
1 | TNF-alpha-induced PGE2 production was evident after 12 h and was associated with a significant TNF-alpha-mediated increase in PGHS-2 immunoreactive protein. | Dinoprostone | tumor necrosis factor | Mus musculus |
2 | TNF-alpha-induced PGE2 production was evident after 12 h and was associated with a significant TNF-alpha-mediated increase in PGHS-2 immunoreactive protein. | Dinoprostone | tumor necrosis factor | Mus musculus |
3 | A specific PGHS-2 inhibitor, NS-398, completely abolished the TNF-alpha-mediated increase in PGE2 production, suggesting that the PGE2 formed in response to TNF-alpha was derived from PGHS-2. | Dinoprostone | tumor necrosis factor | Mus musculus |
4 | A specific PGHS-2 inhibitor, NS-398, completely abolished the TNF-alpha-mediated increase in PGE2 production, suggesting that the PGE2 formed in response to TNF-alpha was derived from PGHS-2. | Dinoprostone | tumor necrosis factor | Mus musculus |
5 | A specific PGHS-2 inhibitor, NS-398, completely abolished the TNF-alpha-mediated increase in PGE2 production, suggesting that the PGE2 formed in response to TNF-alpha was derived from PGHS-2. | Dinoprostone | tumor necrosis factor | Mus musculus |
6 | A specific PGHS-2 inhibitor, NS-398, completely abolished the TNF-alpha-mediated increase in PGE2 production, suggesting that the PGE2 formed in response to TNF-alpha was derived from PGHS-2. | Dinoprostone | tumor necrosis factor | Mus musculus |