Title : All-trans-retinoic acid inhibits Jun N-terminal kinase-dependent signaling pathways.

Pub. Date : 1998 Mar 20

PMID : 9507016






2 Functional Relationships(s)
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1 Furthermore, c-fos expression was inhibited by agonists of retinoic acid receptors (RARs) or retinoid X receptors (RXRs), and suppression of c-fos promoter activity by t-RA was abrogated by treatment with antagonists of RAR-alpha or of all the RXRs. Tretinoin Fos proto-oncogene, AP-1 transcription factor subunit Homo sapiens
2 Furthermore, c-fos expression was inhibited by agonists of retinoic acid receptors (RARs) or retinoid X receptors (RXRs), and suppression of c-fos promoter activity by t-RA was abrogated by treatment with antagonists of RAR-alpha or of all the RXRs. Tretinoin Fos proto-oncogene, AP-1 transcription factor subunit Homo sapiens