Title : Vasoconstrictor-mediated increase in muscle resting thermogenesis is inhibited by membrane-stabilizing agents.

Pub. Date : 1997 Jul

PMID : 9315342






1 Functional Relationships(s)
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1 Similarly, 100 microM (+/-)-propranolol (a dose likely to cause plasma membrane stabilizing effects involving interruption and (or) prevention of action potentials) blocked increases in VO2, lactate, and glycerol efflux by 5 nM angiotensin II (a nonadrenergic vasoconstrictor) with only marginal effects on pressure development. Propranolol angiotensinogen Rattus norvegicus