Title : Role of the stress-activated/c-Jun NH2-terminal protein kinase pathway in the cellular response to adriamycin and other chemotherapeutic drugs.

Pub. Date : 1996 Nov 29

PMID : 8940082






1 Functional Relationships(s)
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Protein Name
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1 Treatment with vinblastine or etoposide (VP-16) also activated JNK, with maximum increases of 6.5- and 4.3-fold, respectively. Etoposide mitogen-activated protein kinase 8 Homo sapiens