Title : A new gastric ulcer model in rats produced by ferrous iron and ascorbic acid injection.

Pub. Date : 1995

PMID : 8536816






4 Functional Relationships(s)
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1 Treatment with superoxide dismutase (SOD, recombinant human CuZnSOD) significantly reduced the size of the ulcers and inhibited the accumulation in lipid peroxides in the gastric mucosa, while treatment with apo-SOD or heat-inactivated SOD did not. Lipid Peroxides superoxide dismutase 1 Homo sapiens
2 Treatment with superoxide dismutase (SOD, recombinant human CuZnSOD) significantly reduced the size of the ulcers and inhibited the accumulation in lipid peroxides in the gastric mucosa, while treatment with apo-SOD or heat-inactivated SOD did not. Lipid Peroxides superoxide dismutase 1 Homo sapiens
3 Treatment with superoxide dismutase (SOD, recombinant human CuZnSOD) significantly reduced the size of the ulcers and inhibited the accumulation in lipid peroxides in the gastric mucosa, while treatment with apo-SOD or heat-inactivated SOD did not. Lipid Peroxides superoxide dismutase 1 Homo sapiens
4 Treatment with superoxide dismutase (SOD, recombinant human CuZnSOD) significantly reduced the size of the ulcers and inhibited the accumulation in lipid peroxides in the gastric mucosa, while treatment with apo-SOD or heat-inactivated SOD did not. Lipid Peroxides superoxide dismutase 1 Homo sapiens