Title : Restoration by bromocriptine of glucocorticoid receptors and glucocorticoid negative feedback on prolactin secretion in estrogen-induced pituitary tumors.

Pub. Date : 1993 Sep

PMID : 8255389






4 Functional Relationships(s)
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1 In controls, serum PRL markedly increased after ether and this effect was blunted by prior dexamethasone (DEX) administration, due to the steroid negative feedback on PRL secretion. Steroids prolactin Rattus norvegicus
2 In controls, serum PRL markedly increased after ether and this effect was blunted by prior dexamethasone (DEX) administration, due to the steroid negative feedback on PRL secretion. Steroids prolactin Rattus norvegicus
3 In this respect, inefficient steroid negative feedback on PRL synthesis due to down-regulation of GCR may contribute to hyperprolactinemia. Steroids prolactin Rattus norvegicus
4 This mechanism is supported from the restoration of GCR and steroid negative feedback on serum PRL by treatment of tumor-bearing rats with BROM. Steroids prolactin Rattus norvegicus