Title : ApoA-IHelsinki (Lys107-->0) associated with reduced HDL cholesterol and LpA-I:A-II deficiency.

Pub. Date : 1995 Sep

PMID : 7670941






1 Functional Relationships(s)
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1 Our data are consistent with the concept that a low HDL cholesterol level in subjects heterozygous for the apoA-IHelsinki mutation (Lys107-->0) having normal LCAT activity is a consequence of decreased concentration of LpA-I:A-II particles and of a smaller size and reduced cholesterol content of HDL particles. Cholesterol NLR family pyrin domain containing 3 Homo sapiens