Title : Negative cross-talk between RelA and the glucocorticoid receptor: a possible mechanism for the antiinflammatory action of glucocorticoids.

Pub. Date : 1995 Apr

PMID : 7659084






1 Functional Relationships(s)
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1 We found that a functional NF-kappa B site in the ICAM-1 promoter, which can be activated by either 12-O-tetradecanoylphorbol-13-acetate or tumor necrosis factor-alpha (TNF alpha), is also the target for glucocorticoids. Tetradecanoylphorbol Acetate intercellular adhesion molecule 1 Homo sapiens