Title : Increased expression of SLC25A1/CIC causes an autistic-like phenotype with altered neuron morphology.

Pub. Date : 2022 Apr 18

PMID : 35203088






2 Functional Relationships(s)
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1 AT-1 overexpressing neurons maintain the cytosolic pool of acetyl-CoA by upregulation of SLC25A1, the mitochondrial citrate/malate antiporter and ATP citrate lyase, which converts cytosolic citrate into acetyl-CoA. Acetyl Coenzyme A ATP citrate lyase Mus musculus
2 AT-1 overexpressing neurons maintain the cytosolic pool of acetyl-CoA by upregulation of SLC25A1, the mitochondrial citrate/malate antiporter and ATP citrate lyase, which converts cytosolic citrate into acetyl-CoA. Acetyl Coenzyme A ATP citrate lyase Mus musculus