Title : Phytic acid-modified CeO2 as Ca2+ inhibitor for a security reversal of tumor drug resistance.

Pub. Date : 2022

PMID : 35126877






4 Functional Relationships(s)
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1 Using doxorubicin (Dox) as a model chemotherapeutic drug, the Ca2+ nano-inhibitors efficiently deprived intracellular excessive free Ca2+, suppressed P-glycoprotein (P-gp) expression and significantly enhanced intracellular drug accumulation in Dox-resistant tumor cells. Doxorubicin ATP binding cassette subfamily B member 1 Homo sapiens
2 Using doxorubicin (Dox) as a model chemotherapeutic drug, the Ca2+ nano-inhibitors efficiently deprived intracellular excessive free Ca2+, suppressed P-glycoprotein (P-gp) expression and significantly enhanced intracellular drug accumulation in Dox-resistant tumor cells. Doxorubicin ATP binding cassette subfamily B member 1 Homo sapiens
3 Using doxorubicin (Dox) as a model chemotherapeutic drug, the Ca2+ nano-inhibitors efficiently deprived intracellular excessive free Ca2+, suppressed P-glycoprotein (P-gp) expression and significantly enhanced intracellular drug accumulation in Dox-resistant tumor cells. Doxorubicin ATP binding cassette subfamily B member 1 Homo sapiens
4 Using doxorubicin (Dox) as a model chemotherapeutic drug, the Ca2+ nano-inhibitors efficiently deprived intracellular excessive free Ca2+, suppressed P-glycoprotein (P-gp) expression and significantly enhanced intracellular drug accumulation in Dox-resistant tumor cells. Doxorubicin ATP binding cassette subfamily B member 1 Homo sapiens