Title : Mutyh deficiency downregulates mitochondrial fusion proteins and causes cardiac dysfunction via α-ketoglutaric acid reduction with oxidative stress.

Pub. Date : 2022 Feb

PMID : 35098839






1 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 MutY homolog (MUTYH), an important protein in base excision repair (BER) system, excises adenine in the nascent strand opposite 8-oxoguanine in template DNA and restores G:C base-pair to maintain the fidelity of DNA replication. Adenine mutY DNA glycosylase Homo sapiens