Title : Baicalin ameliorates 2,4-dinitrochlorobenzene-induced atopic dermatitis-like skin lesions in mice through modulating skin barrier function, gut microbiota and JAK/STAT pathway.

Pub. Date : 2022 Feb

PMID : 34929516






1 Functional Relationships(s)
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1 Pseudo germ-free (GF) DNCB-treated mice receiving fecal microbiota from baicalin donors reduced the dorsal skin thickness and skin EASI score, and inhibited the release of IgE, histamine, TNF-alpha and IL-4 in serum of mice. Dinitrochlorobenzene interleukin 4 Mus musculus