Title : Deficiency of β-arrestin2 alleviates apoptosis through GRP78-ATF6-CHOP signaling pathway in primary Sjögren's syndrome.

Pub. Date : 2021 Dec

PMID : 34710848






2 Functional Relationships(s)
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1 In vitro, we used IFNalpha to stimulate human salivary gland epithelial cells (HSGECs), and the results showed that IFNalpha activated GRP78-ATF6-CHOP apoptosis signaling, decreased cell viability, and induced apoptosis, which were negatively regulated by the ERS inhibitor 4-PBA. 4-phenylbutylamine interferon alpha 1 Homo sapiens
2 In vitro, we used IFNalpha to stimulate human salivary gland epithelial cells (HSGECs), and the results showed that IFNalpha activated GRP78-ATF6-CHOP apoptosis signaling, decreased cell viability, and induced apoptosis, which were negatively regulated by the ERS inhibitor 4-PBA. 4-phenylbutylamine interferon alpha 1 Homo sapiens