Title : YAP-Dependent BiP Induction Is Involved in Nicotine-Mediated Oral Cancer Malignancy.

Pub. Date : 2021 Aug 13

PMID : 34440849






1 Functional Relationships(s)
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1 Mechanistically, we observed that nicotine regulated YAP nuclear translocation and its interaction with TEAD through alpha7-nAChR-Akt signaling, subsequently resulting in increased TEAD occupancy on the HSPA5 promoter and elevated promoter activity. Nicotine cholinergic receptor, nicotinic, alpha polypeptide 7 Mus musculus