Title : Abnormal Mitochondria-Endoplasmic Reticulum Communication Promotes Myocardial Infarction.

Pub. Date : 2021

PMID : 34413791






1 Functional Relationships(s)
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1 The activation of JNK under hypoxic conditions was dependent on overproduction of mitochondrial reactive oxygen species (mtROS) in cardiomyocytes, and mitochondrial division was identified as the upstream inducer of mtROS overproduction. Reactive Oxygen Species mitogen-activated protein kinase 8 Homo sapiens