Title : Disturbed mitochondrial acetylation in accordance with the availability of acetyl groups in hepatocellular carcinoma.

Pub. Date : 2021 Sep

PMID : 34375734






2 Functional Relationships(s)
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1 A hypothetical model of the oncogenic acetylome was proposed that growing demands for histone acetylation in tumor cells would drive the relocalization of acetyl-CoA to the nucleus, which may contribute to the global deacetylation of mitochondrial proteins to support the nuclear acetyl-CoA pool in an ACLY-dependent manner. Acetyl Coenzyme A ATP citrate lyase Homo sapiens
2 A hypothetical model of the oncogenic acetylome was proposed that growing demands for histone acetylation in tumor cells would drive the relocalization of acetyl-CoA to the nucleus, which may contribute to the global deacetylation of mitochondrial proteins to support the nuclear acetyl-CoA pool in an ACLY-dependent manner. Acetyl Coenzyme A ATP citrate lyase Homo sapiens