Title : MicroRNA-494 Regulates Endoplasmic Reticulum Stress in Endothelial Cells.

Pub. Date : 2021

PMID : 34322482






3 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 Pharmacological ER stress inducers (tunicamycin (TCN) and thapsigargin) and hyperglycemia robustly increase the expression of miR-494 in vitro. Tunicamycin microRNA 494 Homo sapiens
2 Pharmacological ER stress inducers (tunicamycin (TCN) and thapsigargin) and hyperglycemia robustly increase the expression of miR-494 in vitro. Tunicamycin microRNA 494 Homo sapiens
3 Surprisingly, miR-494 pretreatment dampens the induction and magnitude of ER stress in response to TCN in endothelial cells and increases cell viability. Tunicamycin microRNA 494 Homo sapiens