Title : Nicotine Enhances Object Recognition Memory via Stimulating α4β2 and α7 Nicotinic Acetylcholine Receptors in the Medial Prefrontal Cortex of Mice.

Pub. Date : 2021

PMID : 34193682






4 Functional Relationships(s)
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1 Intra-mPFC infusion of mecamylamine, a non-selective nicotinic acetylcholine receptor (nAChR) antagonist, 5 min before nicotine administration blocked the effect of nicotine. Nicotine cholinergic receptor, nicotinic, alpha polypeptide 7 Mus musculus
2 Intra-mPFC infusion of mecamylamine, a non-selective nicotinic acetylcholine receptor (nAChR) antagonist, 5 min before nicotine administration blocked the effect of nicotine. Nicotine cholinergic receptor, nicotinic, alpha polypeptide 7 Mus musculus
3 Additionally, intra-mPFC infusion of dihydro-beta-erythroidine, a selective alpha4beta2 nAChR antagonist, or methyllycaconitine, a selective alpha7 nAChR antagonist, significantly suppressed the nicotine-induced object recognition memory enhancement. Nicotine cholinergic receptor, nicotinic, alpha polypeptide 7 Mus musculus
4 Additionally, intra-mPFC infusion of dihydro-beta-erythroidine, a selective alpha4beta2 nAChR antagonist, or methyllycaconitine, a selective alpha7 nAChR antagonist, significantly suppressed the nicotine-induced object recognition memory enhancement. Nicotine cholinergic receptor, nicotinic, alpha polypeptide 7 Mus musculus