Title : Assessment of azithromycin as an anticancer agent for treatment of imatinib sensitive and resistant CML cells.

Pub. Date : 2021 Mar

PMID : 33607534






1 Functional Relationships(s)
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1 Moreover, combination of AZT/IMA suppressed cell viability, induced apoptosis and caspase3/7 activity more effectively and significantly compared to K562R cells treated with only IMA or AZT. Imatinib Mesylate caspase 3 Homo sapiens