Title : Assessment of azithromycin as an anticancer agent for treatment of imatinib sensitive and resistant CML cells.

Pub. Date : 2021 Mar

PMID : 33607534






2 Functional Relationships(s)
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1 RESULTS: AZT treatment alone inhibited cell viability, induced apoptosis and enhanced caspase 3/7 activity in both K562S and high MDR-1 (Pgp) expressing K562R cells. Azithromycin caspase 3 Homo sapiens
2 Moreover, combination of AZT/IMA suppressed cell viability, induced apoptosis and caspase3/7 activity more effectively and significantly compared to K562R cells treated with only IMA or AZT. Azithromycin caspase 3 Homo sapiens