Title : Cellular stress signaling activates type-I IFN response through FOXO3-regulated lamin posttranslational modification.

Pub. Date : 2021 Jan 28

PMID : 33510167






4 Functional Relationships(s)
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1 We show that oxidative stress activates FOXO3 and its transcriptional target glycine-N-methyltransferase (GNMT) whose upregulation triggers depletion of s-adenosylmethionine (SAM), a key co-substrate involved in methyl group transfer reactions. S-Adenosylmethionine glycine N-methyltransferase Homo sapiens
2 We show that oxidative stress activates FOXO3 and its transcriptional target glycine-N-methyltransferase (GNMT) whose upregulation triggers depletion of s-adenosylmethionine (SAM), a key co-substrate involved in methyl group transfer reactions. S-Adenosylmethionine glycine N-methyltransferase Homo sapiens
3 We show that oxidative stress activates FOXO3 and its transcriptional target glycine-N-methyltransferase (GNMT) whose upregulation triggers depletion of s-adenosylmethionine (SAM), a key co-substrate involved in methyl group transfer reactions. S-Adenosylmethionine glycine N-methyltransferase Homo sapiens
4 We show that oxidative stress activates FOXO3 and its transcriptional target glycine-N-methyltransferase (GNMT) whose upregulation triggers depletion of s-adenosylmethionine (SAM), a key co-substrate involved in methyl group transfer reactions. S-Adenosylmethionine glycine N-methyltransferase Homo sapiens