Title : Mulberry leaves extract ameliorates alcohol-induced liver damages through reduction of acetaldehyde toxicity and inhibition of apoptosis caused by oxidative stress signals.

Pub. Date : 2021

PMID : 33390773






2 Functional Relationships(s)
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1 Acute alcohol exposure-induced leukocyte infiltration and pro-inflammation factors, including cyclooxygenase-2 (COX-2), tumor necrosis factor-alpha (TNF-alpha), and interleukin-6 (IL-6), were blocked by MLE in proportion to MLE concentration. Alcohols interleukin 6 Homo sapiens
2 Acute alcohol exposure-induced leukocyte infiltration and pro-inflammation factors, including cyclooxygenase-2 (COX-2), tumor necrosis factor-alpha (TNF-alpha), and interleukin-6 (IL-6), were blocked by MLE in proportion to MLE concentration. Alcohols interleukin 6 Homo sapiens