Title : Endoplasmic reticulum stress in doxorubicin-induced cardiotoxicity may be therapeutically targeted by natural and chemical compounds: A review.

Pub. Date : 2021 Feb

PMID : 33348022






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1 This article reviews several chemical compounds that have been shown to regulate DOX-induced inflammation, apoptosis, and autophagy via inhibition of ER stress signaling pathways, such as the IRE1alpha/ASK1/JNK, IRE1alpha/JNK/Beclin-1, and CHOP pathways. Doxorubicin DNA damage inducible transcript 3 Homo sapiens