Title : MicroRNA-200a represses myocardial infarction-related cell death and inflammation by targeting the Keap1/Nrf2 and β-catenin pathways.

Pub. Date : 2021 Mar-Apr

PMID : 33197602






1 Functional Relationships(s)
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1 CONCLUSION: The results showed that miR-200a expression was inhibited in murine cardiomyocytes due to H2O2 stress in MI cardiac tissues and overexpressed miR-200a could protect the cells from death by regulating the Keap1/Nrf2 and beta-catenin signal transduction pathways. Hydrogen Peroxide nuclear factor, erythroid derived 2, like 2 Mus musculus