Title : Propranolol induced apoptosis and autophagy via the ROS/JNK signaling pathway in Human Ovarian Cancer.

Pub. Date : 2020

PMID : 32922532






3 Functional Relationships(s)
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1 In addition, reactive oxygen species (ROS) increased dramatically after Propranolol treatment and Propranolol activated the phosphorylation of JNK. Reactive Oxygen Species mitogen-activated protein kinase 8 Homo sapiens
2 In addition, reactive oxygen species (ROS) increased dramatically after Propranolol treatment and Propranolol activated the phosphorylation of JNK. Reactive Oxygen Species mitogen-activated protein kinase 8 Homo sapiens
3 In summary, these results suggested that Propranolol induced cell apoptosis and protective autophagy through the ROS/JNK signaling pathway in human ovarian cancer cells. Reactive Oxygen Species mitogen-activated protein kinase 8 Homo sapiens