Title : Catalpol ameliorates doxorubicin-induced inflammation and oxidative stress in H9C2 cells through PPAR-γ activation.

Pub. Date : 2020 Aug

PMID : 32765656






1 Functional Relationships(s)
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1 Catalpol treatment also significantly decreased the expression levels of inflammatory factors (TNF-alpha, IL-1beta and IL-6) in DOX-induced H9C2 cells, which was reversed by transfections with short hairpin RNA targeting PPAR-gamma. catalpol interleukin 1 alpha Rattus norvegicus