Title : MBNL1 reverses the proliferation defect of skeletal muscle satellite cells in myotonic dystrophy type 1 by inhibiting autophagy via the mTOR pathway.

Pub. Date : 2020 Jul 18

PMID : 32683410






1 Functional Relationships(s)
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1 Finally, treatment with rapamycin (an mTOR inhibitor) was shown to abolish the increased proliferation capability of DM1 SSCs due to MBNL1 overexpression. Sirolimus muscleblind like splicing regulator 1 Homo sapiens