Title : Glutamyl-Prolyl-tRNA Synthetase Regulates Proline-Rich Pro-Fibrotic Protein Synthesis During Cardiac Fibrosis.

Pub. Date : 2020 Aug 28

PMID : 32611237






2 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 Inhibition of EPRS using a prolyl-tRNA synthetase (PRS)-specific inhibitor, halofuginone (Halo), significantly decreases translation efficiency of proline-rich collagens in cardiac fibroblasts as well as TGF-beta-activated myofibroblasts. halofuginone transforming growth factor alpha Mus musculus
2 Inhibition of EPRS using a prolyl-tRNA synthetase (PRS)-specific inhibitor, halofuginone (Halo), significantly decreases translation efficiency of proline-rich collagens in cardiac fibroblasts as well as TGF-beta-activated myofibroblasts. halofuginone transforming growth factor alpha Mus musculus