Title : (-)-Epicatechin Modulates Mitochondrial Redox in Vascular Cell Models of Oxidative Stress.

Pub. Date : 2020

PMID : 32587663






4 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 Diabetes-mediated generation of excess reactive oxygen species (ROS) may contribute to vascular dysfunction via damage to mitochondria and regulation of endothelial nitric oxide synthase (eNOS). Reactive Oxygen Species nitric oxide synthase 3 Homo sapiens
2 Diabetes-mediated generation of excess reactive oxygen species (ROS) may contribute to vascular dysfunction via damage to mitochondria and regulation of endothelial nitric oxide synthase (eNOS). Reactive Oxygen Species nitric oxide synthase 3 Homo sapiens
3 Diabetes-mediated generation of excess reactive oxygen species (ROS) may contribute to vascular dysfunction via damage to mitochondria and regulation of endothelial nitric oxide synthase (eNOS). Reactive Oxygen Species nitric oxide synthase 3 Homo sapiens
4 Diabetes-mediated generation of excess reactive oxygen species (ROS) may contribute to vascular dysfunction via damage to mitochondria and regulation of endothelial nitric oxide synthase (eNOS). Reactive Oxygen Species nitric oxide synthase 3 Homo sapiens