Title : Nuclear inclusions of pathogenic ataxin-1 induce oxidative stress and perturb the protein synthesis machinery.

Pub. Date : 2020 May

PMID : 32145456






6 Functional Relationships(s)
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Sentence
Compound Name
Protein Name
Organism
1 Spinocerebellar ataxia type-1 (SCA1) is caused by an abnormally expanded polyglutamine (polyQ) tract in ataxin-1. polyglutamine ataxin 1 Homo sapiens
2 Spinocerebellar ataxia type-1 (SCA1) is caused by an abnormally expanded polyglutamine (polyQ) tract in ataxin-1. polyglutamine ataxin 1 Homo sapiens
3 Spinocerebellar ataxia type-1 (SCA1) is caused by an abnormally expanded polyglutamine (polyQ) tract in ataxin-1. polyglutamine ataxin 1 Homo sapiens
4 Spinocerebellar ataxia type-1 (SCA1) is caused by an abnormally expanded polyglutamine (polyQ) tract in ataxin-1. polyglutamine ataxin 1 Homo sapiens
5 Spinocerebellar ataxia type-1 (SCA1) is caused by an abnormally expanded polyglutamine (polyQ) tract in ataxin-1. polyglutamine ataxin 1 Homo sapiens
6 Spinocerebellar ataxia type-1 (SCA1) is caused by an abnormally expanded polyglutamine (polyQ) tract in ataxin-1. polyglutamine ataxin 1 Homo sapiens